Myocardial Infarction
Measurement and Intervention
(Sprache: Englisch)
Patients currently experiencing acute myocardial infarcts are the beneficiaries of information gathered during the 80 years since this clinical phenomenon was described and the 20 years since treatment in coronary care units was introduced. Physicians have...
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Klappentext zu „Myocardial Infarction “
Patients currently experiencing acute myocardial infarcts are the beneficiaries of information gathered during the 80 years since this clinical phenomenon was described and the 20 years since treatment in coronary care units was introduced. Physicians have gained the ability to minimize inhospital mortality from rhythm disturbances and have gained insight into the importance of optimizing both left ventricular fIlling pressure and outflow resistance in the management of myocar dial failure. Understanding of the pathophysiology of acute myocardial infarcts has matured sufficiently so that now it is possible to consider whether an infarct must evolve to a predetermined size or whether the size could be limited by implementing one or more clinically feasible strategies. Concurrently, it has become evident that patients with acute infarcts are not as 'fragile' as previously supposed, and that they may undergo procedures such as coronary angiography and coronary bypass surgery with acceptable risks. Clinical trials are currently in progress to assess the possible benefit of various interventions for limiting myocardial infarct size. The outcome of these studies may be used to formulate strategies for clinical care of future patients. If the results are positive, community hospitals may undergo changes even more exten sive than those required when they established coronary care units. If the inter ventions are not proven to provide significant advantages over the course of nature, the current concepts of coronary care may be retained. However, such conclusions will be only as valid as the techniques used to measure infarct size.
Inhaltsverzeichnis zu „Myocardial Infarction “
I. Pathophysiology1. Time course of infarct and healing
II. Methods for Determining Infract Size
2. The ECG: QRS change
3. The ECG: the spatial and nonspatial determinants of the extracellularly recorded potential with emphasis on the TQ-ST segment
4. Enzymatic estimation: creatine kinase
5. Enzymatic estimation: confounding effects of blood flow to infarcted myocardium
6. Hemodynamic measurements
7. Contrast ventriculography
8. Radionuclide ventriculography
9. Ultrasound
10. Computerized tomography
11. Regional myocardial blood flow: a model for assessing intervention therapy in the conscious animal
12. Radionuclide perfusion techniques
13. Infarct-avid imaging techniques
14. Postmortem: anatomic quantitation
15. Postmortem: histochemical techniques
III. Interventions for Limiting Infract Size
16. Overview of potential mechanisms
17. Decreasing myocardial energy utilization
18. Increasing coronary blood flow
19. Alterations in the coagulation system
20. Alterations of the metabolic and cellular responses
21. The relationship between coronary angiographic patterns and the effects of infarct limiting interventions
IV. An Overview
22. Current status of measurements and efforts to reduce myocardial infarct size in man
Bibliographische Angaben
- 2011, Softcover reprint of the original 1st ed. 1982., 564 Seiten, Maße: 24,4 cm, Kartoniert (TB), Englisch
- Herausgegeben von Wagner, G.S.
- Verlag: Springer Netherlands
- ISBN-10: 940097454X
- ISBN-13: 9789400974548
Sprache:
Englisch
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